FAS-L, IL-10, and double-negative CD4 CD8 TCR / T cells are reliable markers of autoimmune lymphoproliferative syndrome (ALPS) associated with FAS loss of function

نویسندگان

  • Aude Magerus-Chatinet
  • Marie-Claude Stolzenberg
  • Maria S. Loffredo
  • Bénédicte Neven
  • Catherine Schaffner
  • Nicolas Ducrot
  • Peter D. Arkwright
  • Brigitte Bader-Meunier
  • José Barbot
  • Stéphane Blanche
  • Jean-Laurent Casanova
  • Marianne Debré
  • Alina Ferster
  • Claire Fieschi
  • Benoit Florkin
  • Claire Galambrun
  • Olivier Hermine
  • Olivier Lambotte
  • Eric Solary
  • Caroline Thomas
  • Francoise Le Deist
  • Capucine Picard
  • Alain Fischer
  • Frédéric Rieux-Laucat
چکیده

Aude Magerus-Chatinet,1,2 Marie-Claude Stolzenberg,1,2 Maria S. Loffredo,1,2 Bénédicte Neven,1-3 Catherine Schaffner,1,2 Nicolas Ducrot,1,2 Peter D. Arkwright,4 Brigitte Bader-Meunier,3 José Barbot,5 Stéphane Blanche,3 Jean-Laurent Casanova,2,3,6 Marianne Debré,3 Alina Ferster,7 Claire Fieschi,8 Benoit Florkin,9 Claire Galambrun,10 Olivier Hermine,2-3,11 Olivier Lambotte,12 Eric Solary,13 Caroline Thomas,14 Francoise Le Deist,15 Capucine Picard,2,3,16 Alain Fischer,1-3 and Frédéric Rieux-Laucat1,2

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Abnormally differentiated CD4+ or CD8+ T cells with phenotypic and genetic features of double negative T cells in human Fas deficiency.

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Natural history of autoimmune lymphoproliferative syndrome associated with FAS gene mutations.

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Dominant interfering fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome

Five unrelated children are described with a rare autoimmune lymphoproliferative syndrome (ALPS) characterized by massive nonmalignant lymphadenopathy, autoimmune phenomena, and expanded populations of TCR-CD3+CD4-CD8- lymphocytes. These findings, suggesting a genetic defect in the ability of T lymphocytes to respond to normal immunoregulatory mechanisms, prompted an evaluation of lymphocyte ap...

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تاریخ انتشار 2009